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Dysfunctional ETC, in turn, further enhances ROS production and increases the disruption of the mitochondrial respiratory function and mitochondrial integrity [129]

By interrupting a key proteinprotein interaction that allows senescent cells to persist, FOXO4-DRI offers the potential to selectively eliminate those cells and thus improve tissue function, reduce inflammation, and restore aspects of youthful physiology

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For the system, maximizing GLP-1 therapy value may require investment in care infrastructure, not just medication access

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