LKB1 Polyclonal Antibody, 20ul[BT-AP05039] Cellular Function Assays Defects in GJC2 are the

Defects in GJC2 are the cause of autosomal recessive Pelizaeus-Merzbacher-like disease-1

Its two SH3 domains direct complex formation with proline-rich regions of other proteins

Cyclin dependent kinase 10 encoded by CDK10 belongs to the CDK subfamily of the Ser/Thr protein kinase family

Excellent mixing (200-1500rpm)

and axial skeleton

LKB1 Polyclonal Antibody, 20ul[BT-AP05039] Cellular Function Assays Defects in GJC2 are theSTK11 (serine threonine kinase 11), which encodes a member of the serine threonine kinase family, regulates cell polarity and functions as a tumor suppressor. Mutations in STK11 have been associated with Peutz Jeghers syndrome, an autosomal dominant disorder characterized by the growth of polyps in the gastrointestinal tract, pigmented macules on the skin and mouth, and other neoplasms. Alternate transcriptional splice variants of STK11 have been