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SLC25A39 as well as mitochondrial GSH may also represent promising drug targets for cancer, since manipulating SLC25A39 functioning or mitochondrial GSH in tumor cells may induce selective oxidative stress, leading to cell death

Under oxidative stress, excessive ROS emissions lead to mitochondrial damage, and loss or defects in mitochondrial energy production can enhance metabolic processes in the tricarboxylic acid (TCA) cycle, ETC, exacerbating ROS release and reducing antioxidant enzymes [81,82,83]

Disruption of GH pulsatility has been associated with impaired IGF-1 production, altered lipid metabolism, and reduced tissue regeneration in experimental models

Carnosine Carnosine: endogenous dipeptide with anti-glycation, antioxidant, and pH-buffering properties

doi: 10.3390/ph3010125

What becomes trickier are those drugs that are not administered by IV, but by another route of administration