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10.1038/nature14344 Nature 66 JiangY.MaoC.YangR.YanB.ShiY.LiuX.et al (2017)

3.3.3 Impact of GPX4 on immune-synovial interactions and joint microenvironment The contribution of GPX4 and ferroptosis to OA pathology extends far beyond direct protection of chondrocytes

The damagerelated molecular patterns released by cell death may transmit lipid peroxidation and trigger other cell death patterns

Elevated baseline insulin or insulin resistance may suggest better tirzepatide efficacy

Participants receiving 1 mg per day of AOD 9604 lost approximately 2.6 kg compared to 0.8 kg in the placebo group