KPSH2 Polyclonal Antibody, 20ul Gene Editing Defects in ATG16L1 are a

Defects in ATG16L1 are a cause of susceptibility to inflammatory bowel disease type 10 (IBD10)

ADCK2 participates in ATP and nucleotide binding

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including AMP-morpholidate

This cytokine can block NF-kappa B activity

KPSH2 Polyclonal Antibody, 20ul Gene Editing Defects in ATG16L1 are aCatalytic activity: ATP + a protein = ADP + a phosphoprotein.,Asn 183 is present instead of the conserved Asp which is expected to be an active site residue.,Belongs to the protein kinase superfamily. CAMK Ser Thr protein kinase family.,Contains 1 protein kinase domain.,