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Similarly, the elevation in gastric levels of COX-1, COX-2, PGE1 and PGI2 supports that augmented prostaglandin synthesis has accompanied the elevated activity of COX enzymes during the early phase of healing, when oxidative injury was dominating the ulcerated gastric tissue

Furthermore, GGC itself may be capable of directly acting as an antioxidant by supporting the activity of the enzyme GSH peroxidase [20], which is responsible for eliminating hydrogen peroxide in the RPE [21]

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This process often involves providing medical records and test results to demonstrate the medical necessity of testosterone therapy